President Donald J. Trump has been the subject of withering attacks on his character and fitness for office since announcing his candidacy for president in 2015. The radical left, Democrats and their media allies, and “Never Trump” Republicans say he poses a threat to the presidency, our governing institutions, and our national character. He’s repeatedly accused of being a liar, racist, misogynist and dictator. Stephen Barry and Marc Z. Lieberman, longtime friends with opposite views of Trump, debate his presidency in real time in this book. Through text messages, they highlight the stark divide in how the public views Trump. From Trump’s efforts to ban immigration from dysfunctional Muslim-majority countries, to his plans to build a wall on the Mexican border, to his initial response to the coronavirus pandemic, the authors consider the consequences of his actions—as well as his motives. For those who oppose Trump, the underlying theme is that he should not be permitted to exercise the powers of his office. But are they right? See the arguments for Trump as well as why it’s imperative that he be allowed to serve.
The volumes in this authoritative series present a multidisciplinary approach to modeling and simulation of flows in the cardiovascular and ventilatory systems, especially multiscale modeling and coupled simulations. Volume 5 is devoted to cells, tissues, and organs of the cardiovascular and ventilatory systems with an emphasis on mechanotransduction-based regulation of flow. The blood vessel wall is a living tissue that quickly reacts to loads applied on it by the flowing blood. In any segment of a blood vessel, the endothelial and smooth muscle cells can sense unusual time variations in small-magnitude wall shear stress and large-amplitude wall stretch generated by abnormal hemodynamic stresses. These cells respond with a short-time scale (from seconds to hours) to adapt the vessel caliber. Since such adaptive cell activities can be described using mathematical models, a key objective of this volume is to identify the mesoscopic agents and nanoscopic mediators required to derive adequate mathematical models. The resulting biomathematical models and corresponding simulation software can be incorporated into platforms developed in virtual physiology for improved understanding and training.
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