Levinas and the Greek Heritage shows that throughout his career, Emmanuel Levinas always admired and recognized his profound debt to Plato and to the philosophical tradition he initiated, which have been largely transmitted to us by the Neoplatonists, most notably Plotinus and Proclus. How can we read Otherwise than Being or Beyond Essence in any other way than as some sort of Neoplatonic programme, prolonging Plato's Good "beyond being" of the republic VI, 509b, in the direction of the "other man," the one which in his "nudity" and "fragility," opens for us the horizon of a new humanism? There are many ways by which one can attempt to go over and above Being, not only a Greek way (primordially metaphysical), but also a Biblical way (mainly ethical). One of the interests of Levinas' philosophy is to show us the hidden community - and perhaps unavoidable interdependency - of these two approaches. One Hundred Years of Neoplatonism in France shows that during the Twentieth century a retrieval of Neoplatonism is a powerful hidden feature of French philosophy and theology, of spiritual and institutional life. Beginning with Henri Bergson, it passes by way of figures like Maurice Blondel, A.J. Festugire, Henri de Lubac, Jean Trouillard, Henry Dumry, and culminates with Michel Henry, Pierre Hadot, and Jean-Luc Marion. The book examines the particular character Neoplatonism takes in this retrieval, and traces connections between leading figures within the French and Anglophone worlds.
The volumes in this authoritative series present a multidisciplinary approach to modeling and simulation of flows in the cardiovascular and ventilatory systems, especially multiscale modeling and coupled simulations. Volume 5 is devoted to cells, tissues, and organs of the cardiovascular and ventilatory systems with an emphasis on mechanotransduction-based regulation of flow. The blood vessel wall is a living tissue that quickly reacts to loads applied on it by the flowing blood. In any segment of a blood vessel, the endothelial and smooth muscle cells can sense unusual time variations in small-magnitude wall shear stress and large-amplitude wall stretch generated by abnormal hemodynamic stresses. These cells respond with a short-time scale (from seconds to hours) to adapt the vessel caliber. Since such adaptive cell activities can be described using mathematical models, a key objective of this volume is to identify the mesoscopic agents and nanoscopic mediators required to derive adequate mathematical models. The resulting biomathematical models and corresponding simulation software can be incorporated into platforms developed in virtual physiology for improved understanding and training.
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