Common misconceptions underlie many fears about what causes cancer. Misconceptions about the Causes of Cancer examines the scientific evidence from studies in humans, animal cancer tests, exposures to naturally occurring and synthetic chemicals, and methods used to evaluate and regulate cancer risks. Those wishing to disentangle facts from hype will read about the science showing that: Cancer rates are not soaring in the United States and Canada. Synthetic chemicals at levels found in the environment have not been shown to be an important cause of cancer. Reducing pesticide residues is not an effective way to prevent cancer. Human exposures to potential cancer hazards are not primarily to synthetic chemicals. The toxicology of synthetic chemicals is not different from that of natural chemicals, and natural chemicals make up 99.9% of chemical exposure. High-dose animal cancer tests do not provide enough information to assess human cancer risks at the usual levels of exposure. Pesticides and other synthetic chemicals are not likely to be disrupting hormones. Current regulatory policy of low, hypothetical risks is not effective in advancing public health. Book jacket.
This unique new reference contains the Carcinogenic Potency Database (CPDB), which analyzes results of decades of animal cancer tests, including all Technical Reports of the National Toxicology Program (NTP) and the general published literature. A guide to the literature of animal cancer tests, the CPDB includes references to each published experiment and never-before published analyses. For each of 5,000 long-term experiments on 1,300 chemicals, the user-friendly format includes data on the species, strain, and sex of the test animal; features of experimental protocol such as the route of administration, duration of dosing, dose levels, and duration of the experiment; histopathology and tumor incidence; the shape of the dose-response curve; published author's opinion about the carcinogenicity at each site; and reference to the original publication of the test results. In addition, a measure of carcinogenic potency, the TD50, its statistical significance and confidence limits, are given for each tumor site. An overview is provided of earlier publication updates, such as positivity rates, reproducibility, interspecies extrapolation, and ranking possible carcinogenic hazards. The book also includes a summary of the NTP genetic toxicity test results on 1,500 chemicals, which are referenced to the original publications, including the Salmonella (Ames) test, L5178Y mouse lymphoma cell mutation test, chromosome aberration and sister chromatid exchange tests in cultured Chinese hamster ovary cells, and the sex-linked recessive lethal mutation test in Drosophila melanogaster. An index with chemicals listed by CAS number allows cross referencing between the carcinogenicity and genotoxicity databases, making data easy to find.
This unique new reference contains the Carcinogenic Potency Database (CPDB), which analyzes results of decades of animal cancer tests, including all Technical Reports of the National Toxicology Program (NTP) and the general published literature. A guide to the literature of animal cancer tests, the CPDB includes references to each published experiment and never-before published analyses. For each of 5,000 long-term experiments on 1,300 chemicals, the user-friendly format includes data on the species, strain, and sex of the test animal; features of experimental protocol such as the route of administration, duration of dosing, dose levels, and duration of the experiment; histopathology and tumor incidence; the shape of the dose-response curve; published author's opinion about the carcinogenicity at each site; and reference to the original publication of the test results. In addition, a measure of carcinogenic potency, the TD50, its statistical significance and confidence limits, are given for each tumor site. An overview is provided of earlier publication updates, such as positivity rates, reproducibility, interspecies extrapolation, and ranking possible carcinogenic hazards. The book also includes a summary of the NTP genetic toxicity test results on 1,500 chemicals, which are referenced to the original publications, including the Salmonella (Ames) test, L5178Y mouse lymphoma cell mutation test, chromosome aberration and sister chromatid exchange tests in cultured Chinese hamster ovary cells, and the sex-linked recessive lethal mutation test in Drosophila melanogaster. An index with chemicals listed by CAS number allows cross referencing between the carcinogenicity and genotoxicity databases, making data easy to find.
Common misconceptions underlie many fears about what causes cancer. Misconceptions about the Causes of Cancer examines the scientific evidence from studies in humans, animal cancer tests, exposures to naturally occurring and synthetic chemicals, and methods used to evaluate and regulate cancer risks. Those wishing to disentangle facts from hype will read about the science showing that: Cancer rates are not soaring in the United States and Canada. Synthetic chemicals at levels found in the environment have not been shown to be an important cause of cancer. Reducing pesticide residues is not an effective way to prevent cancer. Human exposures to potential cancer hazards are not primarily to synthetic chemicals. The toxicology of synthetic chemicals is not different from that of natural chemicals, and natural chemicals make up 99.9% of chemical exposure. High-dose animal cancer tests do not provide enough information to assess human cancer risks at the usual levels of exposure. Pesticides and other synthetic chemicals are not likely to be disrupting hormones. Current regulatory policy of low, hypothetical risks is not effective in advancing public health. Book jacket.
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