A fast-moving memoir of twentieth century life, 1931 to 2005, beginning with a Pennsylvania coal region youth, moving through show business, the Army, the Catholic Church, government service, and water supply leadership.
The Catholic Church remains one of the oldest institutions of Western civilization. It continues to withstand attack from without and defection from within. In his revision of American Catholicism, Monsignor Ellis has added a new chapter on the history of the Church since 1956. Here he deals with developments in Catholic education, with the changing relations of the Church to its own members and to society in general, and especially with arguments for and against the ecumenical movement brought about by Vatican Council II. The author gives an updated historical account of the part played by Catholics in both the American Revolution and the Civil War, and of the difficulties within the Church that came with the clash of national interests among Irish, French, and Germans in the nineteenth century. He regards immigration as the key to the increasingly important role of American Catholicism in the nation after 1820. For contemporary America, the author counts among the signs of the mature Church an increase in Church membership, the presence of nine Americans in the College of Cardinals in May, 1967, and the expansion of American effort in Catholic missions throughout the world.
One of the major biomedical triumphs of the post-World War II era was the defmitive demonstration that hypercholesterolemia is a key causative factor in atherosclerosis; that hypercholesterolemia can be effectively treated; and that treatment significantly reduces not only coronary disease mortality but also all cause mortality. Treatment to lower plasma levels of cholesterol - primarily low density lipoprotein (LDL) cholesterol - is now accepted as best medical practice and both physicians and patients are being educated to take aggressive measures to lower LDL. We can confidently look forward to important decreases in the toll of coronary artery disease over the coming decades. However, there is still uncertainty as to the exact mechanisms by which elevated plasma cholesterol and LDL levels initiate and favor the progression of lesions. There is general consensus that one of the earliest responses to hypercholesterolemia is the adhesion of monocytes to aortic endothelial cells followed by their penetration into the subendothelial space, where they differentiate into macrophages. These cells, and also medial smooth muscle cells that have migrated into the subendothelial space, then become loaded with mUltiple, large droplets of cholesterol esters . . . the hallmark of the earliest visible atherosclerotic lesion, the so-called fatty streak. This lesion is the precursor of the more advanced lesions, both in animal models and in humans. Thus the centrality of hypercholesterolemia cannot be overstated. Still, the atherogenic process is complex and evolves over a long period of time.
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